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About Us

 At Age Ex we try to make scientific research more understandable to the general public by analyzing and interpreting research articles related to the benefits of exercise for different chronic age related conditions.


At first glance our audience might appear to be the aging population; which is completely true. However, this blog also targets family members of the elderly to make sure adequate knowledge is transferred even easier. 


Our goal is to broaden people’s understanding about the benefits of exercise in the elderly population and for people to then use this information to consult licensed healthcare professionals to develop a well-rounded healthcare plan.

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The Effect of Exercise on Type 1 Diabetes: More than Meets the Eye

  Pathology :                Insulin is an important player for anabolic reactions in the body. In other words, when there are plenty of nutrients, like after a meal, insulin signaling makes the body use those and fill up the fatty acid and glycogen storages, and also signals for muscle build up (i.e., protein synthesis). Thus, insulin is a crucial endocrine hormone which without it many body functions are impaired. In type 1 diabetes, autoimmune destruction of beta cells takes place and due to loss of beta cell mass, the body cannot make enough insulin or even none at all. How is it bad? Before we go any further, let’s do a simplified run of how insulin works. There is about 4 grams of glucose circulating in the blood of healthy individuals all the time. After a meal or snack, when blood glucose rises, insulin is secreted into the bloodstream. How? Beta cells of the pancreas make insulin which is later picked up by blood circulatin...
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YES!! Physical Activity Can Benefit Those with ALS.

Pathology Amyotrophic lateral sclerosis or ALS is a motor neuron neurodegenerative disease that, like Alzheimer’s, is also associated with cognitive impairments as there are overlaps between ALS and frontotemporal degeneration. The causes for this disease are not exactly known. Although there are known genetic and environmental factors that lead to this disease, the interaction of genetic and environmental exposure can vary individually. You might ask how the interaction between the two leads to ALS. Well, there are certain genes (we carry them all along our lifetime) which are acted on by time and environmental exposure. These continue to have their effects until a certain point in time when the process of neurodegeneration begins and symptoms appear. Clinical presentations are divided into bulbar or spinal-onset and depend on the degree of upper motor neuron or lower motor neuron involvement. In the bulbar onset the disease affects speech and swallowing (dysarthria and dysphagia)...
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Lewy Body Dementia: Can Physical Activity be a Non-pharmaceutical Intervention?

  Pathology: Lewy body Dementia (LBD) is a very complicated disease both in terms of pathology and treatment. This is the one type of disease that has both characteristics of Alzheimer’s disease and Parkinson’s disease. What distinguishes between the types of diseases is the timing of the appearance of symptoms. If symptoms of dementia happen before that of Parkinson’s, the situation is diagnosed as dementia with Lewy bodies or Lewy body dementia. On the contrary, Parkinson’s disease dementia is diagnosed when cognitive impairment happens after Parkinson’s disease. In essence, it is the formation of Lewy bodies and over time the abnormal accumulation of these bodies that leads to the clinical symptoms. These protein deposits often take place in pre-synaptic terminals that are responsible for neurotransmitter release and cycling. Symptoms of LBD can be divided to three levels of cognitive, behavioral, and physical. Cognitive symptoms can begin with presentation of non-amnestic cogni...
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